Cell Proliferation on Titania Layer with In Vitro Apatite Forming Ability

S. Yabe; Kanji Tsuru; Satoshi Hayakawa; Akiyoshi Osaka; Y. Yoshida; K. Suzuki; T. Kuboki

doi:10.4028/www.scientific.net/KEM.330-332.131

Paper Titles

Hydroxyapatite Precursor Phases Identification by X-Ray Diffraction Using Synchrotron Radiation
p.115

Molecular and Crystal Structure Characterization of Calcium-Deficient Apatite
p.119

Studies on Cadmium Uptake by Hydroxyapatite
p.123

Lessening Hydroxyapatite Ceramics Particles Adverse Effects by Serum Modification: A Preliminary In Vitro Study and Mechanism Analysis
p.127

Cell Proliferation on Titania Layer with In Vitro Apatite Forming Ability
p.131

In Vivo Evaluation of Hydrothermally Converted Hydroxyapatite as a Bone Graft Substitute
p.135

Electrohydrodynamic Processing Routes for Bioceramics
p.139

Influence of Solvent Polarity on Synthesis of Beta - Tricalcium Phosphate
p.143

Influence of Hydrothermal Reaction Temperature and pH on Phase Stability of Hydroxyapatite
p.147

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Cell Proliferation on Titania Layer with In Vitro Apatite Forming Ability

Abstract:

Titania layer was fabricated on the titanium substrates with chemical treatment with 20ml or 40ml of hydrogen peroxide solution and subsequent heat treatment at 400°C, coded as CHT20 and CHT40, respectively. CHT20 spontaneously deposited apatite on the surface in a simulated body fluid (SBF), while CHT40 did not. TF-XRD patterns showed that the diffraction intensity of anatase of CHT20 was higher than that of CHT40. It was suggested that the thicker titania layer indicated in vitro apatite forming ability. The cell proliferation of CHT20 and CHT40 were lower than NT and HT. Since the surface of titania layers became hydrophobic after autoclaving, we can suppose that the cell proliferation on CHT20 and CHT40 were lower than NT and HT due to their surface hydrophobicity.